Chapter 20 of 42 · 2818 words · ~14 min read

CHAPTER VII

DISEASES ARISING FROM FAULTY CONFORMATION

A. SAND-CRACK.

_Definition_.--A solution of continuity of the horn of the foot, occurring usually in the wall, and following the direction of the horn fibres.

_Classification_.--It is usual to classify sand-cracks according to--

_(a) Their Position_.--_Toe-crack_ when occurring in the middle line of the horn of the toe, and _quarter-crack_ when occurring in the horn of the quarters.

Sand-crack of the frog and sand-crack of the sole may also each be met with. They are, however, of rare occurrence, and are seldom serious enough to merit special attention.

The toe-crack is met with more often in the hind-foot than in the fore, while the quarter-crack more often than not makes its appearance in the fore-foot, and is there, as a rule, confined to the inner side. The reasons for these positions being so affected we shall deal with when treating of the causes of sand-crack in general. It is interesting to note that the portions of wall known as inside and outside toe are seldom affected.

_(b) Their Length_.--_Complete_ when they extend from the coronary margin of the wall to its wearing edge; _Incomplete_ when not so extensive.

_(c) Their Severity_.--_Simple_ when they occur in the horn only, and do not implicate the sensitive structures beneath; _Complicated_ when deep enough to allow of laceration and subsequent inflammation of the keratogenous membrane. Such complications may vary from a simple inflammation set up by laceration and irritation of the sensitive structures by particles of dirt and grit that have gained entrance through the crack, to other and more serious changes in the shape of the formation of pus, hæmorrhage from the laminal vessels, caries of the os pedis, or the development of a tumour-like growth of horn on the inner surface of the wall known as a keraphyllocele.

_(d) Their Duration_.--_Recent_ when newly formed; _old_ when of long standing.

_(e) Their Starting-point_.--This last distinction we make ourselves, and, referring to cracks of the wall, term them _high_ when commencing from the coronary margin, _low_ when starting from the bearing surface.

_Causes_.--We have already classified sand-crack as a disease arising from faulty conformation. Thus, in just so far as a predisposing build of body may be handed down from parent to offspring, we may regard sand-crack as hereditary. If we do so, however, we must afterwards make up our minds to sharply distinguish between the sand-crack plainly brought about by accidental cause, and that occurring as a result of hereditary evil conformation.

With regard to the latter, we need hardly say that feet with abnormally brittle horn are extremely liable. But with this, as with many other affections of the feet, we shall find it necessary to consider several causes acting in cooperation. In this case, for instance, given the brittle horn, it becomes necessary to further look for exciting causes of its fracture.

We will take conformation first. In the animal with turned-out toes a more than fair share of the body-weight is imposed on the horn of the inner quarter. Here, then, three causes exert their influence together: The horn is brittle; the wall of the inner quarter is thinner than that of the outer; additional weight is imposed upon it. Fracture results.

Take, again, the vice of contracted heels. Here, in the first place, we have a variety of causes tending to bring about the contraction. With the contraction, and its consequent pressure upon the sensitive structures in the region of the quarters and the frog, has arisen a low type of inflammation. The horn of the part has become dry and brittle. The exciting cause of its fracture is found in an excessive day's work upon a hard, dry road, with, perhaps, a suddenly-imposed improper distribution of weight, due to treading upon a loose stone, or a succession of such evil transfers of weight due to travelling upon a road that is rough in its whole extent.

In their turn, too, such defects of the feet as we have mentioned in the last chapter--as, for example, the foot with the pumiced horn, the foot with abnormally upright heels, or that which is upright on one side only, or crooked--each offers a condition which is predisposing to the formation of a sand-crack. In each case it wants but the uneven distribution of the body-weight, which, as a matter of fact, some of these conditions themselves give, to bring about a fracture.

Apart from the predisposition conferred by conformation, must be remembered the simpler predisposing causes leading to brittleness of the hoof. We refer to the after-effects of poulticing, the moving from pasture to stable, the emigration from a damp to a dry climate, or the alternate changes from damp to dry in a temperate region. Each may have a deteriorating influence upon the horn, rendering it liable to the condition we are describing. Excessive dampness alone, especially when the animal is called upon to labour at the drawing of heavy loads upon a rough road, is not infrequently a cause. In this case the wet, together with the constant friction of the sharp materials of which the road is made, serves to destroy the varnish-like periople. The wet gains access to the inner structures of the wall, the agglutination of the horn fibres is weakened, and fissures begin to appear.

Other causes of sand-crack are purely accidental. An animal at fast work over-reaches. The secretion of horn at the injured coronet is interfered with, a diminished supply at an isolated spot being the result. From this point grows down a fissure in the wall.

An injury of the same character may also be sustained in various other ways--treads from other animals when working in pairs, accidental wounding with the stable-fork, blows of any kind, or a self-inflicted tread with the calkin of an opposite foot--each with the same result.

So far as causation is concerned, toe-crack stands in a class almost by itself. It is met with nearly always in a heavy animal in the hind-foot, and is directly attributable to the force exerted in starting a heavy load.

Unskilful shoeing also plays a part in the causation of sand-crack. Removal of the periople by excessive rasping of the wall is most certainly a predisposing cause. Cracks, or their starting-points, may also be caused by using too wide a shoe, or by the use of nails too large in the shank. Also, they may arise from unskilful fitting of the toe-clip, especially in the hind-foot of a heavy animal. It must be admitted, however, that the part shoeing plays in the causation of sand-crack is not a large one; far more depends upon the state of the horn and the animal's conformation than upon the exciting cause.

So far, our observations on the causes of sand-crack have referred to that form occurring in the wall. Sand-crack of the sole or frog we have already said is but seldom met with, and then it is always in connection with some exceptionally deteriorated quality of the horn, as in the case of badly pumiced feet, or occurs as a result of direct injury. Extensive slit-like cuts in this region, when deep enough to lacerate the keratogenous membrane, are sometimes followed by the growth of a fissure in the horn, and what might almost be termed a permanent sand-crack results. Such cuts may be occasioned by sharp flints, broken glass, or other sharp objects picked up on the road, or may result from the animal treading on the toe-clip of a partially cast shoe.

_Symptoms_.--In every case the fissure, or evidence of its commencement, is a diagnostic symptom. It is well to remember, however, that this may be easily overlooked, especially when the crack is one commencing at the coronary margin. The reason is this: Sand-cracks in this position often commence in the wall proper, and not in the periople. They may, in fact, be first observed as a fine separation of the horn fibres immediately beneath the perioplic covering. A crack of this description may even show hæmorrhage, and have been in existence for some time, without the periople itself showing any lesion whatever. Thus, unless lameness is present, or a more than specially keen search is directed to the parts in question, the sand-crack goes undiscovered, until of greater dimensions.

Further, the fissure may be hidden, either accidentally or of set purpose. It may be covered by the hair, filled in and covered over with mud, or intentionally concealed by being 'stopped' with an artificial horn, with wax, or with gutta-percha, or, as is more common, be hidden by the lavish application of a greasy hoof-dressing.

In this latter connection it is well to warn the veterinary surgeon, especially the beginner, when examining for soundness, to be keenly critical before passing an animal who is presented with feet smothered with tar and grease or any other dressing. More especially should this warning be heeded when examining any of the heavier breeds of animal with an abundance of hair about the coronet.

Referring again to the search for the crack, it is well to know that with toe-crack the fissure is the more readily seen when the foot is lifted from the ground. With quarter-crack, on the other hand, the fissure is wider, and consequently the easier detected with the foot bearing weight.

Although commencing in the insidious manner we have described, the lesion is not thus often seen by the veterinary surgeon. Usually, the animal with sand-crack is brought for his inspection when lameness has arisen from it. In this case the cause for the lameness will reveal itself in the crack, which is now too large to escape observation. The coronet is hot and tender to the touch, and a sensation of warmth is sometimes conveyed to the hand by the horn of the surrounding parts of the wall. It is hardly necessary to say that, with accompanying conditions such as these, the sand-crack is a _deep_ one.

Where the lameness is but slight, we may attribute it almost solely to the pain occasioned by the mere wounding of the keratogenous membrane, and to no very extensive inflammatory changes therein. By some authorities this is said to be due to the pinching of the sensitive structures between the edges of the fissure in the horny covering. In our opinion, however, pinching does not occur unless inflammatory exudation into the sensitive structures adjoining the crack has led to sufficient swelling to cause them to protrude. In other words, the movements of the horny box, communicating themselves to the structures beneath, and so occasioning movement in the wounded keratogenous membrane, are quite sufficient to give rise to the lameness without actual pinching of the structures implicated.

The severity of the lameness will vary with the rapidity of the gait, and with the character of the road upon which the animal is made to travel. For instance, many animals in which the lameness is imperceptible at a walk become 'dead' lame at a fast trot. It is sufficiently explained when one remembers the greater movements of expansion and contraction of the posterior parts of the wall brought about by the increase in the rate of progression. The same animal, too, will go distinctly more lame upon a hard than upon a soft surface.

In like manner the lameness from toe-crack also varies in degree with the rate of progression and the character of the travelling, though not to such a noticeable extent as in the lameness from quarter-crack. A greater variation may in this case be brought about by moving the animal on ascending and descending ground. Descending an incline, with a more than ordinary share of the body-weight thus thrown upon the heels, the lameness is most marked. The reason would appear to be that the greater expansion of the wall of the heels thus brought about leads to a proportionate contraction of the wall at the toe, especially at the edges of the crack, thus causing undue pressure upon the exact spot of the wound in the sensitive structures. Ascending--the weight in this case transferred from the posterior to the anterior portion of the foot--the expansion of the heels becomes a contraction, with a corresponding lessening of the contraction at the toe and a distinct decrease in the lameness.

In the case of a deep but recent crack there is always more or less hæmorrhage. This favours risk of infection of the lesion with pus-forming organisms, and so leads to a more or less pronounced lameness, a degree of swelling, heat and tenderness in the coronet above, and a certain amount of surgical fever.

The acute symptoms subdued, but the fissure still remaining, gives us the crack we have classified as 'old.' This may in every case be distinguished from a more recent lesion by the amount of thickening of the overhanging coronet, and the presence of an increased quantity of sub-coronary horn in the region immediately about the crack. The previous inflammatory changes in the adjoining sensitive structures have here led to an increased secretion of horn, and a greater or less deposition of inflammatory connective tissue in the wounded coronary cushion.

Sand-crack of the toe always follows the direction of the horn fibres. That of the quarter, however, may on occasion run a course that is somewhat zigzag, first following the direction of the horn fibres for a short distance, then travelling in a horizontal direction, and finally continuing its course again in a line with the horn fibres, commonly at a point posterior to that at which it commenced.

In a quarter-crack that is old, and when contraction of the heels exists (which in this case it usually does), then will often be found overlapping of the edges of the crack. The expansion of the wall brought about when the body-weight is on the heels, cannot, by reason of the break in it, continue itself anterior to the crack. As a consequence, repeated expansion of the wall posterior to the crack, with the portions anterior to it in a state of enforced quiescence, leads in time to the posterior edge of the crack coming to lie over that of the anterior.

_Complications_.--The first complication likely to arise in a case of sand-crack is that attending simple laceration of the sensitive structures in a _deep_ lesion. With the laceration all the phenomena of a repairing inflammation make their appearance. As a result, there is more or less heat according to the degree of inflammatory hyperæmia, swelling according to the amount of inflammatory exudate, and pain according to the amount of pressure the two foregoing bring to bear on the nerves in the inflamed area.

A second and more serious complication is the greater inflammation set up by the introduction into the crack of foreign substances. Small portions of gravel and flint, both by the irritation set up by their friction and by the infection they carry in with the dirt surrounding them, are responsible for the mischief.

When, from direct communication with the blood-stream, due to extensive hæmorrhage, bacteria from the outside gain entrance, this simple inflammation is further complicated by the formation of pus, or a limited gangrene of the keratogenous membrane.

In cases of great severity the gangrene of the keratogenous membrane spreads until the deeper structures are involved. We then get a necrosis (in the case of toe-crack) of the extensor pedis, and sometimes caries of the os pedis.

In like manner the necrotic changes occurring under these circumstances may invade the deeper structures in the region of quarter-crack. As a result of this, we may have the starting-point of suppurating corn, or necrosis of the lateral cartilage--in other words, cartilaginous quittor.

Commonly accompanying quarter-crack is the condition of contracted heels and atrophied frog. Sometimes described as a complication of sand-crack, it appears to us more rational to rather regard the sand-crack as a result or complication of the vice of contraction.

The overlapping of the edges of the crack before referred to occasionally gives rise to the condition known as false quittor. A probe or a director passed beneath the overhanging ledge of horn reveals sometimes a fissure of 1 inch or considerably more in depth, and quittor is diagnosed. A careful paring away of the overhanging horn, however, reveals the true state of affairs, and exposes to view the original cause of the mischief--a simple fissure in the wall.

A serious complication--one fortunately met with but rarely--is that of keraphyllocele. This is a tumour-like growth of horn, varying in size from the thickness of an ordinary quill pen to that of one's middle finger, growing down from the coronary cushion, and attached to the inner side of the wall of the hoof. With this lameness is always present, and more or less deformity of the hoof results. This condition will be found described at greater length in